Yeast and Rickets

Rickets

Rickets in Tehranian Children: Occurance of Chronic Sinusitis and Giardiosis in Late Rickets

SUMMARY A review of out patients diagnosed with rickets during last one and half year revealed 135 cases of rickets among totally 1550 patients refered to our pediatric infectious clinics. Diagnosis were made by the clinical, biochemical, radiological and pathological findings. All the patients were treated by Vit D as needed, follow up examinations were done at least for 6m. We could not see ...

199 - Osteomalacia and rickets*

1997 Vitamin D is converted in the liver by a vitamin D-25-hydroxylase (25-OHase) to form the major circulating form of vitamin D, 25-hydroxyvitamin D [25(OH)D]. There have been at least four different 25-OHases identified both in mitochrondia and in microsomes. 25(OH)D is, however, biologically inert and requires hydroxylation in the kidneys on carbon 1 by the CYP27B mitrochondrial enzyme 25-h...

Rickets and soil strontium.

The subjects of this study were children aged 6-60 months living in villages in the Ulas Health Region, Sivas. The villages were divided into two groups according to the amount of strontium in the soil: region 1, > 350 ppm, 650 children; region 2, < 350 ppm, 1596 children. Overall, the prevalence of one or more clinical signs of rickets was 22.9%. The prevalence in region 1 was 31.5% and that i...

Hypophosphatemic rickets and osteomalacia.

The hypophosphatemic conditions that interfere in bone mineralization comprise many hereditary or acquired diseases, all of them sharing the same pathophysiologic mechanism: reduction in the phosphate reabsorption by the renal tubuli. This process leads to chronic hyperphosphaturia and hypophosphatemia, associated with inappropriately normal or low levels of calcitriol, causing osteomalacia or ...